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Dissecting cellular senescence and SASP in Drosophila | Inflammation and  Regeneration | Full Text
Dissecting cellular senescence and SASP in Drosophila | Inflammation and Regeneration | Full Text

Ras/ERK-signalling promotes tRNA synthesis and growth via the RNA  polymerase III repressor Maf1 in Drosophila | PLOS Genetics
Ras/ERK-signalling promotes tRNA synthesis and growth via the RNA polymerase III repressor Maf1 in Drosophila | PLOS Genetics

Addgene: LZRS ERTm RasV12
Addgene: LZRS ERTm RasV12

IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis  Model Fueled by Tumor/Host Interactions
IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis Model Fueled by Tumor/Host Interactions

Interaction between RasV12 and scribbled clones induces tumour growth and  invasion | Nature
Interaction between RasV12 and scribbled clones induces tumour growth and invasion | Nature

JNK and Yorkie drive tumor malignancy by inducing L-amino acid transporter  1 in Drosophila | PLOS Genetics
JNK and Yorkie drive tumor malignancy by inducing L-amino acid transporter 1 in Drosophila | PLOS Genetics

Dissemination of RasV12-transformed cells requires the mechanosensitive  channel Piezo | Nature Communications
Dissemination of RasV12-transformed cells requires the mechanosensitive channel Piezo | Nature Communications

EPLIN is a crucial regulator for extrusion of RasV12- transformed cells
EPLIN is a crucial regulator for extrusion of RasV12- transformed cells

Dissemination of RasV12-transformed cells requires the mechanosensitive  channel Piezo | Nature Communications
Dissemination of RasV12-transformed cells requires the mechanosensitive channel Piezo | Nature Communications

pQCXP CMV/TO RasV12 (w364-1) - Addgene
pQCXP CMV/TO RasV12 (w364-1) - Addgene

FBP17-mediated finger-like membrane protrusions in cell competition between  normal and RasV12-transformed cells - ScienceDirect
FBP17-mediated finger-like membrane protrusions in cell competition between normal and RasV12-transformed cells - ScienceDirect

Report Loss of Cell Polarity Drives Tumor Growth and Invasion through JNK  Activation in Drosophila
Report Loss of Cell Polarity Drives Tumor Growth and Invasion through JNK Activation in Drosophila

Loss of Misshapen (msn) synergizes with oncogenic Ras (RasV12) to... |  Download Scientific Diagram
Loss of Misshapen (msn) synergizes with oncogenic Ras (RasV12) to... | Download Scientific Diagram

Ets21C is required for RasV12 dlgRNAi tumors.: Confocal images of (a–c)...  | Download Scientific Diagram
Ets21C is required for RasV12 dlgRNAi tumors.: Confocal images of (a–c)... | Download Scientific Diagram

Sequential oncogenic mutations influence cell competition - ScienceDirect
Sequential oncogenic mutations influence cell competition - ScienceDirect

Fig. 2. Salivary glands from RasV12-expressing larvae produce MMP1, release  tissue fragments into the hemolymph and express apoptotic markers.Salivary.  - ppt download
Fig. 2. Salivary glands from RasV12-expressing larvae produce MMP1, release tissue fragments into the hemolymph and express apoptotic markers.Salivary. - ppt download

Summary of experimental findings. In K-Ras V12 mutant cell lines, |  Download Scientific Diagram
Summary of experimental findings. In K-Ras V12 mutant cell lines, | Download Scientific Diagram

An Ectopic Network of Transcription Factors Regulated by Hippo Signaling  Drives Growth and Invasion of a Malignant Tumor Model - ScienceDirect
An Ectopic Network of Transcription Factors Regulated by Hippo Signaling Drives Growth and Invasion of a Malignant Tumor Model - ScienceDirect

Research Plan | Kyoto University Medical School Department of Molecular  Oncology
Research Plan | Kyoto University Medical School Department of Molecular Oncology

Mutations in the Drosophila tricellular junction protein M6 synergize with  RasV12 to induce apical cell delamination and invasion | PNAS
Mutations in the Drosophila tricellular junction protein M6 synergize with RasV12 to induce apical cell delamination and invasion | PNAS

Biomedicines | Free Full-Text | Ha-RasV12-Induced Multilayer Cellular  Aggregates Is Mediated by Rac1 Activation Rather Than YAP Activation
Biomedicines | Free Full-Text | Ha-RasV12-Induced Multilayer Cellular Aggregates Is Mediated by Rac1 Activation Rather Than YAP Activation

RasV12-driven tumour formation depends on the setting of an EGF/EFGR... |  Download Scientific Diagram
RasV12-driven tumour formation depends on the setting of an EGF/EFGR... | Download Scientific Diagram

IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis  Model Fueled by Tumor/Host Interactions
IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis Model Fueled by Tumor/Host Interactions

Endothelial k-RasV12 Expression Induces Capillary Deficiency Attributable  to Marked Tube Network Expansion Coupled to Reduced Pericytes and Basement  Membranes | Arteriosclerosis, Thrombosis, and Vascular Biology
Endothelial k-RasV12 Expression Induces Capillary Deficiency Attributable to Marked Tube Network Expansion Coupled to Reduced Pericytes and Basement Membranes | Arteriosclerosis, Thrombosis, and Vascular Biology

IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis  Model Fueled by Tumor/Host Interactions
IJMS | Free Full-Text | RasV12; scrib−/− Tumors: A Cooperative Oncogenesis Model Fueled by Tumor/Host Interactions

Mutations in the Drosophila tricellular junction protein M6 synergize with  RasV12 to induce apical cell delamination and invasion | PNAS
Mutations in the Drosophila tricellular junction protein M6 synergize with RasV12 to induce apical cell delamination and invasion | PNAS